![]() If this respiratory acidosis was chronic we would expect that the kidneys would have generated more HCO 3– to compensate, which would have resulted in an increased BE.The base excess is within normal limits as there has been no significant change in the amount of HCO 3–.The HCO 3– is normal, so the metabolic system is not contributing to the acidosis and also isn’t compensating for the respiratory acidosis, suggesting that this is an acute derangement.In the context of low PaO 2, a raised PaCO 2 suggests the patient has type 2 respiratory failure.In this case, the PaCO 2 is raised significantly and this is likely to be the cause of the acidosis.You should then note that the pH reveals an acidosis and assess the CO 2 to see if it is contributing to the acidosis (↑CO 2). ![]() ![]() The PaO 2 is low, so we know the patient is in respiratory failure, however, we don’t yet know what type. ![]()
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